Mouse models of acute promyelocytic leukemia
Current Opinion in Hematology, ISSN: 1065-6251, Vol: 8, Issue: 4, Page: 206-211
2001
- 23Citations
- 10Captures
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Example: if you select the 1-year option for an article published in 2019 and a metric category shows 90%, that means that the article or review is performing better than 90% of the other articles/reviews published in that journal in 2019. If you select the 3-year option for the same article published in 2019 and the metric category shows 90%, that means that the article or review is performing better than 90% of the other articles/reviews published in that journal in 2019, 2018 and 2017.
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Metrics Details
- Citations23
- Citation Indexes23
- 23
- CrossRef18
- Captures10
- Readers10
- 10
Review Description
Translocations involving a variety of fusion partners, such as promyelocytic leukemia gene, promyelocytic leukemia zinc finger, nucleophosmin, nuclear matrix protein, and signal transducer and activator of transcription protein 5B, with the retinoic acid receptor α gene are commonly associated with development of acute promyelocytic leukemia. Through the development of transgenic mouse models, some retinoic acid receptor α translocation fusion proteins have been shown to be capable of initiating acute promyelocytic leukemia development, and dictate the leukemias' responsiveness to retinoic acid. Transgenic mouse models also have identified the influence of reciprocal translocation fusion proteins on acute promyelocytic leukemia development, and have demonstrated that additional mutations can contribute to the development of acute promyelocytic leukemia. In this review, the authors summarize current mouse models of acute promyelocytic leukemia and describe current knowledge about additional genetic alterations that occur during development of acute promyelocytic leukemia in the mouse. © 2001 Lippincott Williams & Wilkins, Inc.
Bibliographic Details
http://www.scopus.com/inward/record.url?partnerID=HzOxMe3b&scp=0034891006&origin=inward; http://dx.doi.org/10.1097/00062752-200107000-00005; http://www.ncbi.nlm.nih.gov/pubmed/11561157; http://journals.lww.com/00062752-200107000-00005; https://dx.doi.org/10.1097/00062752-200107000-00005; https://journals.lww.com/co-hematology/Abstract/2001/07000/Mouse_models_of_acute_promyelocytic_leukemia.5.aspx
Ovid Technologies (Wolters Kluwer Health)
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