Expression of gp34 (OX40 ligand) and OX40 on human T cell clones
Japanese Journal of Cancer Research, ISSN: 0910-5050, Vol: 92, Issue: 4, Page: 377-382
2001
- 37Citations
- 20Captures
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Example: if you select the 1-year option for an article published in 2019 and a metric category shows 90%, that means that the article or review is performing better than 90% of the other articles/reviews published in that journal in 2019. If you select the 3-year option for the same article published in 2019 and the metric category shows 90%, that means that the article or review is performing better than 90% of the other articles/reviews published in that journal in 2019, 2018 and 2017.
Citation Benchmarking is provided by Scopus and SciVal and is different from the metrics context provided by PlumX Metrics.
Metrics Details
- Citations37
- Citation Indexes37
- 37
- CrossRef33
- Captures20
- Readers20
- 20
Article Description
gp34, Which we previously cloned, is a ligand of OX40 (CD134), a costimulatory molecule involved in T cell activation. To elucidate the role of human OX40/OX40L interaction, we examined the expression of gp34 (OX40L) and OX40 in normal human hematopoietic cells by using flow cytometry. OX40 expression is observed on activated T cells, while OX40L is expressed in antigen-presenting cells. However, cytotoxic T lymphocyte (CTL) clones specific for Epstein-Barr virus (EBV)-transformed autologous lymphoblastic cell lines (LCLs) induced both OX40 and OX40L expression after antigen or T cell receptor (TCR) stimulation. This study suggests a possible function of OX40L/OX40, through T cell-T cell interaction, in the reactivation of memory T cells in an autocrine manner, with implications for the pathogenesis of viral infections and neoplasms.
Bibliographic Details
http://www.scopus.com/inward/record.url?partnerID=HzOxMe3b&scp=0035025030&origin=inward; http://dx.doi.org/10.1111/j.1349-7006.2001.tb01105.x; http://www.ncbi.nlm.nih.gov/pubmed/11346458; https://onlinelibrary.wiley.com/doi/10.1111/j.1349-7006.2001.tb01105.x; https://dx.doi.org/10.1111/j.1349-7006.2001.tb01105.x
Wiley
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