Inhibition of intestinal cell proliferation by villous cell extract
Gut, ISSN: 0017-5749, Vol: 28, Issue: SUPPL.1, Page: 109-115
1987
- 9Citations
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Example: if you select the 1-year option for an article published in 2019 and a metric category shows 90%, that means that the article or review is performing better than 90% of the other articles/reviews published in that journal in 2019. If you select the 3-year option for the same article published in 2019 and the metric category shows 90%, that means that the article or review is performing better than 90% of the other articles/reviews published in that journal in 2019, 2018 and 2017.
Citation Benchmarking is provided by Scopus and SciVal and is different from the metrics context provided by PlumX Metrics.
Metrics Details
- Citations9
- Citation Indexes9
- CrossRef9
Article Description
In order to verify the hypothesis that intestinal cell proliferation is controlled by a mitotic inhibitor, extracts of villous epithelial cells from different species were analysed to study their effect on the proliferation of various intestinal cells. Villous extracts from rat and rabbit strongly and reversibly inhibited cell division and DNA synthesis in a rat intestinal epithelial cell line and a primary culture of rabbit intestinal epithelial cells. This non-cytotoxic, tissue specific but not species specific factor is present in both villous cells and crypt cells, with the highest concentrations occurring in the superficial epithelial cells. Assay of a partial purification of this factor showed that it has a molecular weight of approximately 190 000 daltons.
Bibliographic Details
http://www.scopus.com/inward/record.url?partnerID=HzOxMe3b&scp=0023504721&origin=inward; http://dx.doi.org/10.1136/gut.28.suppl.109; http://www.ncbi.nlm.nih.gov/pubmed/3692299; https://gut.bmj.com/lookup/doi/10.1136/gut.28.Suppl.109; http://gut.bmj.com/cgi/doi/10.1136/gut.28.Suppl.109; https://syndication.highwire.org/content/doi/10.1136/gut.28.Suppl.109; https://dx.doi.org/10.1136/gut.28.suppl.109; https://gut.bmj.com/content/28/supplement/109
BMJ
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