Gastrin and somatostatin in Helicobacter pylon infected antral mucosa
Gut, ISSN: 0017-5749, Vol: 35, Issue: 5, Page: 615-618
1994
- 72Citations
- 15Captures
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Example: if you select the 1-year option for an article published in 2019 and a metric category shows 90%, that means that the article or review is performing better than 90% of the other articles/reviews published in that journal in 2019. If you select the 3-year option for the same article published in 2019 and the metric category shows 90%, that means that the article or review is performing better than 90% of the other articles/reviews published in that journal in 2019, 2018 and 2017.
Citation Benchmarking is provided by Scopus and SciVal and is different from the metrics context provided by PlumX Metrics.
Metrics Details
- Citations72
- Citation Indexes72
- 72
- CrossRef47
- Captures15
- Readers15
- 15
Article Description
Helicobacter pylori infection is associated with increased meal stimulated gastrin secretion, but the reason for this is unknown. Sequence specific radioimmunoassays were used to measure the concentration of α-amidated gastrin, the total progastrin product, and somatostatin in biopsy specimens of human antral mucosa. The antral concentrations of α-amidated gastrin and of total progastrin products were significantly higher in H pylori infected patients than in those not infected by this organism. In contrast, the antral somatostatin concentration was significantly decreased in infected patients. Progastrin processing, determined by gel chromatography, seemed unaffected by H pylori infection. The results suggest that the finding of increased gastrin secretion from the antral G cells in H pylori infected patients may be a result of reduced inhibition of G-cell secretion by somatostatin.
Bibliographic Details
http://www.scopus.com/inward/record.url?partnerID=HzOxMe3b&scp=0028358763&origin=inward; http://www.ncbi.nlm.nih.gov/pubmed/7911115; http://dx.doi.org/10.1136/gut.35.5.615; https://gut.bmj.com/lookup/doi/10.1136/gut.35.5.615; https://dx.doi.org/10.1136/gut.35.5.615; https://gut.bmj.com/content/35/5/615
BMJ
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