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Metabolic profiling of triple-negative breast cancer cells reveals metabolic vulnerabilities.

Cancer & metabolism, ISSN: 2049-3002, Vol: 5, Issue: 1, Page: 6
2017
  • 81
    Citations
  • 0
    Usage
  • 214
    Captures
  • 12
    Mentions
  • 0
    Social Media
Metric Options:   Counts1 Year3 Year

Metrics Details

  • Citations
    81
  • Captures
    214
  • Mentions
    12
    • News Mentions
      11
      • 11
    • Blog Mentions
      1
      • 1

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Article Description

Among breast cancers, the triple-negative breast cancer (TNBC) subtype has the worst prognosis with no approved targeted therapies and only standard chemotherapy as the backbone of systemic therapy. Unique metabolic changes in cancer progression provide innovative therapeutic opportunities. The receptor tyrosine kinases (RTKs) epidermal growth factor receptor (EGFR), and MET receptor are highly expressed in TNBC, making both promising therapeutic targets. RTK signaling profoundly alters cellular metabolism by increasing glucose consumption and subsequently diverting glucose carbon sources into metabolic pathways necessary to support the tumorigenesis. Therefore, detailed metabolic profiles of TNBC subtypes and their response to tyrosine kinase inhibitors may identify therapeutic sensitivities.

Bibliographic Details

Lanning, Nathan J; Castle, Joshua P; Singh, Simar J; Leon, Andre N; Tovar, Elizabeth A; Sanghera, Amandeep; MacKeigan, Jeffrey P; Filipp, Fabian V; Graveel, Carrie R

Springer Science and Business Media LLC

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