Wnt/β-Catenin signaling pathway in hepatocellular carcinoma: pathogenic role and therapeutic target
Frontiers in Oncology, ISSN: 2234-943X, Vol: 14, Page: 1367364
2024
- 11Citations
- 23Captures
- 1Mentions
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Example: if you select the 1-year option for an article published in 2019 and a metric category shows 90%, that means that the article or review is performing better than 90% of the other articles/reviews published in that journal in 2019. If you select the 3-year option for the same article published in 2019 and the metric category shows 90%, that means that the article or review is performing better than 90% of the other articles/reviews published in that journal in 2019, 2018 and 2017.
Citation Benchmarking is provided by Scopus and SciVal and is different from the metrics context provided by PlumX Metrics.
Metrics Details
- Citations11
- Citation Indexes11
- 11
- Captures23
- Readers23
- 23
- Mentions1
- News Mentions1
- News1
Most Recent News
Second Hospital of Lanzhou University Researchers Provide New Study Findings on Liver Cancer (Wnt/b-Catenin signaling pathway in hepatocellular carcinoma: pathogenic role and therapeutic target)
2024 APR 16 (NewsRx) -- By a News Reporter-Staff News Editor at NewsRx Drug Daily -- Investigators publish new report on liver cancer. According to
Review Description
Hepatocellular carcinoma (HCC) is the most common primary malignant liver tumor and one of the leading causes of cancer-related deaths worldwide. The Wnt/β-Catenin signaling pathway is a highly conserved pathway involved in several biological processes, including the improper regulation that leads to the tumorigenesis and progression of cancer. New studies have found that abnormal activation of the Wnt/β-Catenin signaling pathway is a major cause of HCC tumorigenesis, progression, and resistance to therapy. New perspectives and approaches to treating HCC will arise from understanding this pathway. This article offers a thorough analysis of the Wnt/β-Catenin signaling pathway’s function and its therapeutic implications in HCC.
Bibliographic Details
http://www.scopus.com/inward/record.url?partnerID=HzOxMe3b&scp=85190377548&origin=inward; http://dx.doi.org/10.3389/fonc.2024.1367364; http://www.ncbi.nlm.nih.gov/pubmed/38634048; https://www.frontiersin.org/articles/10.3389/fonc.2024.1367364/full; https://dx.doi.org/10.3389/fonc.2024.1367364; https://www.frontiersin.org/journals/oncology/articles/10.3389/fonc.2024.1367364/full
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