Macrophage Dysfunction in Autoimmune Rheumatic Diseases and Atherosclerosis
International Journal of Molecular Sciences, ISSN: 1422-0067, Vol: 23, Issue: 9
2022
- 18Citations
- 20Captures
- 1Mentions
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Example: if you select the 1-year option for an article published in 2019 and a metric category shows 90%, that means that the article or review is performing better than 90% of the other articles/reviews published in that journal in 2019. If you select the 3-year option for the same article published in 2019 and the metric category shows 90%, that means that the article or review is performing better than 90% of the other articles/reviews published in that journal in 2019, 2018 and 2017.
Citation Benchmarking is provided by Scopus and SciVal and is different from the metrics context provided by PlumX Metrics.
Metrics Details
- Citations18
- Citation Indexes18
- 18
- CrossRef12
- Captures20
- Readers20
- 20
- Mentions1
- Blog Mentions1
- Blog1
Most Recent Blog
IJMS, Vol. 23, Pages 4513: Macrophage Dysfunction in Autoimmune Rheumatic Diseases and Atherosclerosis
IJMS, Vol. 23, Pages 4513: Macrophage Dysfunction in Autoimmune Rheumatic Diseases and Atherosclerosis International Journal of Molecular Sciences doi: 10.3390/ijms23094513 Authors: Elena V. Gerasimova Tatiana
Review Description
One of the problems of modern medical science is cardiovascular pathology caused by atherosclerotic vascular lesions in patients with autoimmune rheumatic diseases (ARDs). The similar-ity between the mechanisms of the immunopathogenesis of ARD and chronic low-grade inflammation in atherosclerosis draws attention. According to modern concepts, chronic inflammation associated with uncontrolled activation of both innate and acquired immunity plays a fundamental role in all stages of ARDs and atherosclerotic processes. Macrophage monocytes play an important role among the numerous immune cells and mediators involved in the immunopathogenesis of both ARDs and atherosclerosis. An imbalance between M1-like and M2-like macrophages is considered one of the causes of ARDs. The study of a key pathogenetic factor in the development of autoimmune and atherosclerotic inflammation-activated monocyte/macrophages will deepen the knowledge of chronic inflammation pathogenesis.
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