(n-3) Fatty Acids Alleviate Adipose Tissue Inflammation and Insulin Resistance: Mechanistic Insights
Advances in Nutrition, ISSN: 2161-8313, Vol: 2, Issue: 4, Page: 304-316
2011
- 234Citations
- 319Captures
- 2Mentions
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Example: if you select the 1-year option for an article published in 2019 and a metric category shows 90%, that means that the article or review is performing better than 90% of the other articles/reviews published in that journal in 2019. If you select the 3-year option for the same article published in 2019 and the metric category shows 90%, that means that the article or review is performing better than 90% of the other articles/reviews published in that journal in 2019, 2018 and 2017.
Citation Benchmarking is provided by Scopus and SciVal and is different from the metrics context provided by PlumX Metrics.
Metrics Details
- Citations234
- Citation Indexes234
- 234
- CrossRef203
- Captures319
- Readers319
- 319
- Mentions2
- News Mentions2
- News2
Most Recent News
Impact of Lipids on Insulin Resistance: Insights from Human and Animal Studies
Introduction Insulin resistance (IR) is a complex pathological condition that has become a focal point in studies related to metabolic diseases such as type 2
Review Description
Obesity is associated with the metabolic syndrome, a significant risk factor for developing type 2 diabetes and cardiovascular diseases. Chronic low-grade inflammation occurring in the adipose tissue of obese individuals is causally linked to the pathogenesis of insulin resistance and the metabolic syndrome. Although the exact trigger of this inflammatory process is unknown, adipose tissue hypoxia, endoplasmic reticular stress, and saturated fatty acid–mediated activation of innate immune processes have been identified as important processes in these disorders. Furthermore, macrophages and T lymphocytes have important roles in orchestrating this immune process. Although energy restriction leading to weight loss is the primary dietary intervention to reverse these obesity-associated metabolic disorders, other interventions targeted at alleviating adipose tissue inflammation have not been explored in detail. In this regard, (n-3) PUFA of marine origin both prevent and reverse high-fat-diet–induced adipose tissue inflammation and insulin resistance in rodents. We provide an update on the pathogenesis of adipose tissue inflammation and insulin resistance in obesity and discuss potential mechanisms by which (n-3) PUFA prevent and reverse these changes and the implications in human health.
Bibliographic Details
http://www.sciencedirect.com/science/article/pii/S2161831322009371; http://dx.doi.org/10.3945/an.111.000505; http://www.scopus.com/inward/record.url?partnerID=HzOxMe3b&scp=84655174598&origin=inward; http://www.ncbi.nlm.nih.gov/pubmed/22332072; https://linkinghub.elsevier.com/retrieve/pii/S2161831322009371; https://dx.doi.org/10.3945/an.111.000505
Elsevier BV
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