Cerebral cortex synaptic heavy mitochondria may represent the oldest synaptic mitochondrial population: Biochemical heterogeneity and effects of L-acetylcarnitine
Journal of Bioenergetics and Biomembranes, ISSN: 0145-479X, Vol: 32, Issue: 2, Page: 163-173
2000
- 20Citations
- 10Captures
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Example: if you select the 1-year option for an article published in 2019 and a metric category shows 90%, that means that the article or review is performing better than 90% of the other articles/reviews published in that journal in 2019. If you select the 3-year option for the same article published in 2019 and the metric category shows 90%, that means that the article or review is performing better than 90% of the other articles/reviews published in that journal in 2019, 2018 and 2017.
Citation Benchmarking is provided by Scopus and SciVal and is different from the metrics context provided by PlumX Metrics.
Metrics Details
- Citations20
- Citation Indexes20
- 20
- CrossRef11
- Captures10
- Readers10
- 10
Article Description
The microheterogeneous nature of intrasynaptic mitochondria has been demonstrated and is widely accepted. However, evidence is still lacking about the role played by the different intrasynaptic mitochondrial subpopulations. The data obtained support the hypothesis that 'heavy' mitochondria could represent old mitochondrial populations: in fact, in addition to the well known impairment of typical mitochondrial functions, they possess the highest levels of hydroperoxides and their fatty acids pattern is completely modified. The qualitative and quantitative fatty acid modifications suffered by these organelles deeply altered their protein/lipid ratio, thus modifying their mode of action. The present work also collects a large body of evidence that a subchronic L-acetylcarnitine treatment in 28 days does not structurally affect both nonsynaptic and intrasynaptic mitochondria of normal rat in a 'steady-state' metabolic condition.
Bibliographic Details
http://www.scopus.com/inward/record.url?partnerID=HzOxMe3b&scp=0033865202&origin=inward; http://dx.doi.org/10.1023/a:1005559930210; http://www.ncbi.nlm.nih.gov/pubmed/11768749; http://link.springer.com/10.1023/A:1005559930210; http://dx.doi.org/10.1023/a%3A1005559930210; https://dx.doi.org/10.1023/a%3A1005559930210; https://link.springer.com/article/10.1023/A:1005559930210
Springer Nature
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